Peptide library / LL-37

LL-37

LL-37 is a 37-amino-acid human cathelicidin peptide involved in innate immune defense and inflammatory signaling.

Peptide & blend guide

The short version

Why people discuss it
Discussed for antimicrobial, immune and skin/wound-related goals; its effects are not uniformly anti-inflammatory. Community interest does not establish benefit or a suitable treatment.
What it is
LL-37 is a 37-amino-acid human cathelicidin peptide involved in innate immune defense and inflammatory signaling.
Biggest caution
An endogenous defense role does not establish that systemic synthetic LL-37 is beneficial or safe. Its inflammatory and antimicrobial effects depend on concentration, tissue and experimental setting.
Stack context
Related profiles reflect adjacent community topics or biological context. They do not establish combination benefit, compatibility or a validated regimen.

Dose and frequency reference

Reference only — not a recommendation. Amounts describe source-specific community examples, selected study regimens or the named product label. They do not establish safety, effectiveness, product identity, or a suitable dose for any person. Never convert mg, mcg, or IU into syringe “units” without the exact product concentration.

Sources report different amounts or schedules; the reference below is a named example, not a consensus.

Subcutaneous injection

Community reference · Not clinical guidance
Amount
100–200 mcg
Frequency
Once daily
Duration / phase
2–4 weeks

Community pattern; human studies used topical formulations Reference only; this does not establish safety, effectiveness or individual suitability.

Reference source · reviewed 2026-09-12
Compare reported schedules

Saved source material reviewed 2026-09-27. Multiple pages from one publisher count as one source family; agreement does not establish what most people use. SC means subcutaneous; IM intramuscular; IP intraperitoneal.

Evidence & sources · Community source profile

AxonRep synthesizes community discussion signals from its internal research corpus. Individual creator sources remain attributed to the synthesis; public reference sites used in the latest comparison are linked separately. The synthesis is not clinical evidence.

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